Posted by admin on January 29, 2011 under Herbal | 
Negative Pine person is over-conscientious, never tries to do anything wrong, never injures the feelings of others, is a vegetarian (usually), and is always conscious of his guilt and therefore remains unhappy all the time. He is very apologetic in his every-day life, never asks for anything as a matter of right, but seeks it as a matter of favour.
Because of his feeling that he is always in the wrong, he develops inferiority complex. He sets a very high standard for himself and may have to over-work and strain to do better in order to improve his work and character. If the over-strain adversely affects his health and he is unable to continue his effort, he still blames himself for the ensuing disability.
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Posted by admin on January 23, 2011 under Gastrointestinal |
A Healthy Bowel Movement
This should be bulky, soft and mid-brown in colour; it should smell very little and disintegrate easily in the water of the lavatory.
A Constipated Bowel Movement
Because it has lost more water it is dark and compacted; it can also be streaked with mucus or blood. Blood in the stool must always be investigated even if you feel it is the result of straining to pass the hard faeces. Sometimes this opens up a crack (fissure) around the anus or causes a pile (haemorrhoid) to bleed.
Haemorrhoids
These varicose veins of the rectum are often the butt of music hall jokes, but any sufferer will tell you there is not a lot to laugh about; it is a very painful condition. Local anaesthetic creams and suppositories are on free sale at the pharmacy or can be prescribed by your doctor. They can make life a lot more bearable, but you cannot be really comfortable until you are no longer passing a hard stool. Your doctor will decide whether any medical treatment such as tying off the veins is necessary. The old remedy for piles was to scrape a clove of garlic to release the juice and then put it in the rectum. The effect is probably similar to eating a crushed clove; a fierce heat followed by a soothing warmth.
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Posted by admin on January 15, 2011 under Diabetes |
Type 2 diabetes results in genetically susceptible people from insulin resistance and diminished insulin secretion by the pancreas. Loss of first-phase insulin secretion after a glucose stimulus is the first recognizable pancreatic functional defect. Postnutrient hyperglycemia occurs, and the pancreas secretes an excess of insulin in an attempt to return plasma glucose to or toward normal. In this phase of type 2 diabetes, impaired glucose tolerance is usually demonstrated. Over time, insulin output by the pancreas diminishes, and at the time of diagnosis by fasting hyperglycemia, insulin secretion is approximately 50% of normal. With increasing duration of type 2 diabetes, further diminution of insulin secretion occurs, hepatic glucose output increases, and insulin resistance of muscle, fat, and liver is accentuated by “glucose toxicity.”
This sequence of events can be viewed as occurring in four stages. In stage 1, diminished first-phase insulin secretion and impaired glucose tolerance may be managed by oral drugs that stimulate an immediate insulin release. Insulin resistance can be addressed by an intensive diet and exercise program and by insulin-sensitizing drugs. As the disease progresses, combination therapy that addresses the dual issues of diminished insulin secretion and insulin resistance are indicated. Combination therapy with insulin stimulators (meglitinides or sulfonylureas) and insulin sensitizers (metformin, thiazolidinediones) is needed. Triple oral therapy with an insulin stimulator, a thiazolidinedione to increase insulin sensitivity, and metformin to inhibit hepatic glucose output may be effective. Some physicians advocate adding an alpha glucosidase inhibitor to blunt the absorption of dietary carbohydrates. Thus, serial addition of many oral antidiabetic agents to address the diabetic state is feasible. The final step is insulin therapy, which can be used as basal replacement with continued oral agent therapy or as the sole pharmacologic agent. Guidelines for glycemic regulation are the same as in type 1 diabetes: the goal for fasting blood glucose is 80-120 mg/dl. Action should be taken for HbA1 c > 8%, with a goal of < 7%.
In type 2 diabetes, hyperglycemia appears to be a major contributor to the progression of retinopathy, albuminuria, and neuropathy. In this respect, the main cause of the so-called “microvascular” complications of type 2 diabetes is comparable to type 1 diabetes. In both cases, prospective randomized trials have provided strong support for a policy of lowering HbAlc to as close to normal as safely possible. For every 1% lowering of an elevated HbAlc, there is about a 25% decrease in the risk for progression of diabetic retinopathy.
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